Abstract: Workplace bullying represents an increasingly recognized occupational hazard with consequences extending far beyond immediate psychological distress. Emerging neuroscientific evidence suggests that sustained interpersonal harassment at work may fundamentally alter brain structure and accelerate biological aging processes, potentially increasing vulnerability to dementia and cognitive decline in later life. This article synthesizes neuroimaging research, longitudinal epidemiological studies, and cellular aging research to examine the pathways linking chronic workplace abuse to long-term neurological health outcomes. Findings reveal measurable hippocampal atrophy, gray matter reduction in stress-processing regions, and telomere shortening among bullying victims—biological markers associated with accelerated cognitive aging. These convergent findings support reframing workplace bullying from an individual employment concern to a significant public health issue requiring organizational-level intervention and policy reform. Evidence-based organizational responses are examined across healthcare, financial services, manufacturing, and public sector contexts, emphasizing prevention, early intervention, and systemic culture change as essential components of cognitive health protection in working populations.
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